Myasthenia Natural Diet

Thymus Removal  CIDPUSA.org

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Removal of the thymus gland is widely practiced as long-term therapy for MG. Most patients improve for a period and some may continue improving, while others soon deteriorate again. Josephson reported the complete failure of nutritional and manganese therapy in MG patients who had their thymus removed. Some researchers believe that the great variability of thymectomy outcomes is due to so-called accessory thymuses or pockets of thymus tissue that may be present in the neck area. These will often be sufficient to maintain a reasonable manganese metabolism and, with this, enable an eventual recovery. On the other hand, if all thymus tissue has been removed, then a full recovery may not be possible. However, there is cautious optimism that even then, a holistic approach can still lead to considerable improvement and to some regrowth of any remaining traces of thymus tissue. Also, the removal of the thyroid makes a cure more difficult, as one of Josephson's case histories shows. In addition to enlargement and tumors of the thymus, MG patients frequently have problems with other endocrine glands such as the thyroid, pituitary, and adrenal glands. There is a close relationship between the thymus and the thyroid in that hyperthyroidism generally leads to myasthenia gravis or muscular debility, as well as to hyperplasia of the thymus. As the disease progresses, most or all of the endocrine glands, organs, and metabolic functions tend to deteriorate. This, then, requires in addition to manganese therapy individualized support with a wide range of nutrients and remedies as well as a diet of highest quality.

Thymus & Manganese

 Another set of problems may arise if the disease is due to chemical poisoning. This happened to Simon Kelly . He had developed MG once before, apparently due to extensively working with oil paints in a confined space. Six years later he had another stressful period during which he painted his house and burned off old paint. Not only did he develop MG a second time, but his blood became very alkaline and his red blood cells "looked like sea urchins," shriveled, black, and full of spikes. He also believes that a high consumption of soymilk contributed to his condition by causing intestinal inflammation and diarrhea. After an odyssey of orthodox and alternative treatments, he had his first real improvement during a short period on manganese supplements, and then continued to improve further with Buteyko-type breathing to reduce the alkalinity of his blood. He also used some wheat grass juice. However, his best improvement came after several months of stagnation when he tried a second lot of manganese. His eyes were better than they had been for many years, and the strength of his legs improved tremendously. It is now believed that in this case the poisoning of the energy-producing mitochondria caused an acute deficiency of metabolic acids, especially citric acid, in addition to the leaching of potassium from the poisoned cells. This is like developing chronic fatigue syndrome in addition to MG and may have contributed as much to his weakness as the MG itself. In such cases, clearly the highest quality of support is required.  


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